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It is not uncommon for a woman to be upset or self-conscious about the size of her breasts. Breast enlargement surgery continues to evolve, and this procedure becomes increasingly safer and yields more predictable results. Consequently, an increasing number of women are taking advantage of breast enlargement surgery. Many women do not consider breast enlargement surgery because they think only of the potential side effects. Women worry that the implants will break, increase their risk of breast cancer, or they will not be able to breast feed when they have children. Although these problems may have presented a small risk in the past, breast enlargement has evolved to the point where these risks are virtually eliminated. “It is true that, in the past, it was not uncommon for breast implants to break,” comments Winter Park, Florida cosmetic surgeon Dr. Scott Greenberg, “however, the number of these occurrences are greatly exaggerated over time and, in effect, breast enlargement is generally believed to be a lot more risky than it actually is.” Women use breast enlargement to enhance the contour of their body, to balance breasts that are unequal sizes, to correct a decrease in breast volume following pregnancy, or for reconstructive purposes following breast surgery. Dr. Greenberg adds, “Breast enlargement does more than just increase the size of a woman’s bust line. Many women are self-conscious when their breasts are uneven or if one or both breasts have been removed as a result of cancer treatment. Breast enlargement surgery aims to give these women a better self-image.” Breast implants are inserted through a small incision. The location of this incision depends on the woman’s preference, anatomy, and the surgeon’s recommendation. The implants are either composed of a silicone gel or saline. Both types of implants are FDA approved, however gel implants have not yet been released by the FDA for use in breast augmentation. prosolution penis enargement pills penis enargement testimonials penis enlarement system penis enlarement patch penis enargement before and after permanent pnis enlargement magna rx plus penile enlargment exercise

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In looking through my previous email conversations, I must say I've probably been asked all sorts of questions from my male visitors. I thought it might be interesting to share some of those questions with you simply because if your a man, I'd bet you either have these same exact questions...or are about to ask them to a somebody like me. I'm young will an enlargement program work for me? Most younger men pride themselves on their sexual conquests. They have the ability to engage in sex frequently during a 24 hour period. Conversely, older men can usually only engage in sex one time, but that length of time is of a longer duration. I started this answer with that information to prove a point and set a standard. Younger men have poor sexual lasting skills which is what an enlargement course should provide. Good enlargement programs provide not only exercises to increase size, but have also insured those exercises they use also promote a longer love making time duration. Does it matter if I'm circumcised or not? Whether or not a foreskin is present doesn't matter concerning an enlargement program. If it is a problem, then most likely the exercise itself has been poorly designed or layed out by the site operator. I'm old, will it work for me? Similar to my first question, just the opposite is true. Older men do have a tendency to last longer in bed but their erectile function and penis size have really taken a hit by the passing years. A good enlargement program is able to adapt its exercises to the older gentleman, which insures enhanced erection strength, as well as increased size. Does anything come in the mail? The good penis enlargement programs are completely online with their programs. My program for instance, also offers men the ability to utilize a test to measure their own testosterone levels in the privacy of their home...but by and large, the better enlargement programs themselves are completely online. How long does it take? I've seen some exercise programs developed by individuals that if you followed it exactly, and this is no exaggeration...you'd be doing penis exercises for nearly 5 hours per day. I don't know about anyone else...but that's just way too long and I've got a life! Good programs take anywhere from 15-20 minutes per day. Excellent programs take around ten minutes per day. Perhaps I've been able to answer some questions you might have had about penis enlargement programs...if not, please feel free to visit my website and contact me with them. pnis enlargement photo free pennis enlargement technique pro solution pill male pnis enlargement do penis enlargement pill really work penis enargement pump does magna rx work cheap penis enlargment pills penile enlargement procedure

It might benefit all men to know and understand that erection weakness, as well as premature ejaculation are essentially coded messages from your body needing to be deciphered and understood. The statistics show, and is supported by my findings through contacting members of my penis enlargement exercise website, that most of those suffering from premature ejaculation are actually younger men, who have less control over their bodies and less experience than those of an older man. In fact, erectile dysfunction and premature ejaculation were the most frequently reported sexual concerns according to 9536 men who connected with a male health helpline. While the diagnosis and treatment of the sometimes deep-rooted psychological factors manifesting themselves in the form of premature ejaculation are beyond the scope of this article, what is the scope of this article is the physical treatment of the condition. Erectile dysfunction and premature ejaculation are the most common male sexual disorders and each may have various degrees of severity. Some cases of premature ejaculation are so severe, the man will reach orgasm as soon as his penis makes vaginal contact. This is because two major causes of premature ejaculation are over-eagerness and anxiety. It seems clear that a lot of men who suffer from premature ejaculation are embarrassed to talk about it, even with their doctors. Some of the physical causes of premature ejaculation are diabetes, a tight foreskin, and disorders of the nervous system. Depression may also lead to erectile dysfunction by creating a loss of sex drive, or by the medications used for treatment of the condition. As with other types of sexual dysfunction, there has never been consensus about what exactly constitutes rapid or premature ejaculation. A Possible Treatment Researchers at the University of Minnesota, have shown that the drug dapoxetine is a safe and effective drug treatment for premature ejaculation. In addition a University of Alberta researcher is very close to finding a possible additional promising treatment for premature ejaculation. Until Dapoxetine will be approved by FDA, men need to know there does exist an online program available for possible premature ejaculation treatment, and it's available at my website. If however, you are looking for brick-and-morter treatment options, those possibilities are: Therapy Counseling Medication Combination of these methods. Hypnosis has also proven very effective in the treatment of premature ejaculation in a very small number of men. Many common 'solutions' to prevent premature ejaculation are completely counterproductive. As sexual disorders like premature ejaculation are becoming common around the globe, the Universities will continue to churn out possible medications to treat it with. What they've overlooked, is that most men who suffer with premature ejaculation are actually just in need of a little reprogramming...and a penis exercise program. buy penis enlargement pills top rated pnis enlargement pills magna rx pill penis enlargement traction device penile enlargment before and after penis elargement surgeries penis enlargment pic before and after vimax penis pill penile enlargement procedure

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penis enlagement pills product penis enlagement tool penis enlarement pills review cheap penis enargement penis enlarement product penis enlargment review penis enlargment stretcher pnis enlargement picture penile enlargement procedure

Now, you may ask yourself, what's the connection between arousal and the g spot? Well, the g spot is best found and stimulated when you are completely turned on. If you try to stimulate it before it can be hard to find, and stimulation can be uncomfortable or even painful. The best time to explore the g spot is when your partner is so hot she’s begging you to be inside her. To help you get to that place quickly and easily, we’re focusing this article on getting you both highly aroused - quickly and easily. Speaking of arousal, did you know the most important part of the body for arousal is ... the mind? Yes, its not the clitoris, the penis, or any erogenous zone. Its the mind. About 75% of arousal comes from the mind (especially for women). So, guys, I’ve created a sample evening to use with your partner, and worked in many small techniques to build arousal. I suggest you read this over, completely, and then plan an evening with your partner this week and follow the steps: 1) when you wake up, tell your partner that you love her, and then share two or three things you enjoy about being with her. When you are sharing this, focus on speaking from your root ... you may notice often your energy is in your head, or your throat. Use your breathe, and your awareness to speak from your sex. Speak with heart, and with emotion. be real! 2) tell your partner that you’ve planned a super sexy night for her, and that you have a surprise for her as well. (DO NOT TELL HER WHAT IT IS!) This will let your partner know that tonight is not a normal night. It will also make her think about it all day. This is what you want. 3) don’t spend much time with your partner that day. be out, be busy. its important to have physical seperation, to build a bit of distance. 4) however, make a point to call her and say that you love her and are looking forward to going out later that night. by now, she’ll likely be telling her friends how strange you’re acting! : ) When you are out that night, do whatever you would normally do: cook-in, go out to a nice restaurant, have wine, cocktails, etc. 5) However, when you first greet her, make a point to hug/kiss her with love and intensity. Also, create an excuse to whisper something (anything) in her ear, and let your breath linger on her neck. 6) Then, throughout the evening, create excuses to massage her shoulders, touch her arm or leg, be close to her, etc. Now, you can do all these small techniques things at any time, on any day. (In fact, I recommend you do them all the time!). I’m just framing them over an evening date to make them easier to teach you. Its likely by now that she’ll ask you about your surprise ... don’t tell her what it is. Keep the suspense and mystery. Feel free to be playful – but don’t tell her. Now, its after dinner, you’re coming back home. Tell her that the first part of the surprise is that you’ve planned a special evening once you get back. Say, I’m going to give you a cocktail (or a glass of wine, etc.) when we get home and go upstairs and prepare. This makes her even more curious, and lets her know you’re taking care of everything. Women generally find this super sexy. Go to your bedroom, and set things up. You’re going to be doing some massage and sexual exercises with your partner. Prepare for that, and then invite your partner into the space. Now, in this part of the evening, you’re going to experiment with some simple exercises that build attraction, arousal and intimacy. Again, you can use these techniques any time, together or separate. I’ve just put them into an evening activity as a way to teach them (it also makes for a great night of fun!) 1) tell your partner that you’ve designed an evening to help create intimacy and arousal. that neither of you will be orgasming tonight – that’s its about loving each other, and getting hot and sexy – without the pressure of orgasm. (you heard me – don’t have an orgasm ... take away the predicable path ... you’ll get much more hot for each other, and its good practice. trust me! 2) take a few minutes to share a bit about what you appreciate and admire about each other. we often don’t do this with our partners. share 2 or 3 things each. 3) now, take a few minutes, to look into your partners left eye. sit quietly, be present with each other, feel your partner. if you mind wanders bring it back. this is a small exercise to build presence and intimacy. it will likely feel quite weird in the beginning. do it anyway! 4) if it seems appropriate share a bit about how that ways, and try it again 5) now, take 5 minutes to kiss each other. pretend you’ve just met. explore each other. kiss as if that’s all you are going to do tonight, not as an excuse to get to have sex. explore her lips, tongue, neck, ears, chin, throat, etc. 6) you will likely get bored and want to move on. don’t! i’ve done workshops where you have to kiss 1,000 times differently. be creative. push through the normal patterns. we are retraining how you approach sex. slowing you down. re- awakening your senses. building creative muscle. 7) after this, lay your partner down, and put a blindfold on her. (if you don’t have one, you can use a scarf, or something else) 8) take 10 or 15 minutes to touch your partners body. NOT HER CLITORUS OR VAGINA. enjoy yourself. if you are having fun, you will be much more present, and amazingly enough, it will feel much better to her. experiment with different kinds of touch: hard, fast, slow, soft, circular, fingernails, pads of fingers, feather, silk, tongue, lips, cold breathe, hot breathe, something warm, something cold, etc. track your partner’s response. what you are doing should be turning her on. if its not, try something different, and check in with her 9) never feel bad about requesting, giving or receiving feedback. you can’t know everything, so don’t expect it of yourself. 10) now, at this point in the evening, you can start to innovate. you can either have you partner lay you down and do the same thing to you. you can start to involve her yoni and clitoris. you can branch into massage, oral sex, sex, etc. be creative. Remember, do not have an orgasm tonight. Use the evening to enjoy each other. Revel in each other’s taste, smell’s, and passion. Love and be loved. Find the freedom that comes from not trying to get somewhere.