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1. WHAT ARE SEXUALLY TRANSMITTED DISEASES (STDS)? Sexually transmitted diseases are diseases that can be passed from person to person through sexual contact. In this case sexual contact means penis-vagina penetration, oral sex which is sexual contact using the mouth, and insertion of the penis into the rectum which is anal sex. Some of these diseases may be transmitted by exchange of sexual fluids such as semen or vaginal discharge. Some of the STD's result in open sores, and it can be spread by contact with skin of someone else. There are also ways to transmit these diseases in a non sexual way, an infected pregnant woman can either give it to her baby during pregnancy, or when the baby is being delivered. Drug abusers can transmit the disease through sharing hypodermic needles that have been used by an infected person. The seriousness of STD's varies, some are cured easily by drugs, others need a combination of treatments and drugs, whilst others have no cure, and the only option is treatment. 2. WHAT ARE SEXUALLY TRANSMITTED IINFECTIONS (STI's)? Any infection that is usually passed through sexual contact. 3. ARE THE TWO WORDS INTERCHANGEABLE? Fifteen years ago both these categories came under one name Venereal Disease (VD). To distinguish between them they were separated into infections (STI) and diseases(STD). Infection means that a germ, bacteria, parasite or virus is present in the body. An infected person does not necessarily have any symptoms, which means that they do not usually feel ill..A disease is any abnormal condition of the body or mind that causes discomfort, dysfunction, or distress, in other words your body tells you that you are unwell. This means that STI covers a wider range than the term STD. STD refers only to infections that are causing problems. Because most of the time, people don't know they are infected with an STI until they start showing symptoms of disease, the AIDS Resource Center uses the term STD, even though the term STI is also appropriate in many cases. Let's see if we can simply un-muddy the waters here. Genital herpes has two states when the blisters are present and when they are absent. When they are present they are causing symptoms, ie the blisters, at this stage it is an STD, and it is that this stage that the infection is most likely to be spread to another person.When the blisters are absent then there are no symptoms, and this is then an STI, and the likelihood of an infection is reduced. However HIV can be an infection, in the sense that there may be no symptoms, when they develop symptoms then they have AIDS which is an STD. However it is important to remember that HIV infection can be spread at any time. 4.WHAT IS THE RELATIONSHIP BETWEEN STD'S AND HIV? A person who is already infected with STD, has a higher risk of contracting HIV if they have unprotected sex, without a condom. This risk is greater if the STD causes open genital sores, as these wounds provide a break in the skin which enables the HIV infection to enter the blood stream. STDs that can cause genital ulcers include genital herpes, syphilis, chancre, gonorrhoea, trichomoniasis, and scabies. 5.WHY IS IT DIFFICULT TO RECOGNIZE THAT YOU MAY HAVE A STD? First of all the majority of the people with STD have no immediate symptoms and when they do have them it can be misleading to diagnose as the symptoms can be confused with non sexual diseases. Please note that this applies much more to women than men. 6. WHAT ARE THE MOST COMMON SYMPTOMS FOR WOMEN WHEN THEY ARE SUFFERING FROM AN STD? unusual or bad-smelling vaginal discharge, severe itching or burning in the genital area, unusual bleeding, pain in the pelvic region, pain during sex, rashes on the genitals, open sores or warts on the genital area, and/or recurrent urinary tract infections. 7. WHAT ARE THE MOST COMMON SYMPTOMS FOR MEN WHEN THEY ARE SUFFERING FROM AN STD? In men, the most common symptoms of STD are: pain when urinating, open sores or warts on the genital are genital rash discharge from the penis, and/or pain in the scrotum/testicles. 8. WHAT ARE THE OTHER SYMPTOMS NOT CONNECTED TO THE GENITALS? The following symptoms are present in both men and women: discharge from the anus, swelling of the groin, jaundice (yellowing of the skin and whites of the eyes), oral thrush (white tongue), arthritis, sores or bumps in and around the mouth, and generalized rashes. 9 CAN I CONTRACT STI AS A RESULT OF MUTUAL MASTERBATION? Yes you can and listed below are some examples: Bacterial Vaginosis Cytomegalovirus (CMV) Herpes Simplex Human Papilloma Virus (HPV, Warts) Pubic Lice Scabies 10. CAN I PREVENT GETTING STI or STD? The only foolproof way is abstinence from sex. A condom merely reduces the risk, and it must be used every time, before any sexual fluids are exchanged. penis enlargement excercises free penis elargement video plus vig rx penis enlarement surgeries discount vigrx penis elargement review pennis enlargement exercise penis enlagement surgery

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Your penis is determined by genetics. (According to the National Center for Genome Research at the National Institute of Health. "penis size like height, weight, and general build, probably comes from both mom and pop.) So I guess you can go ahead and blame your mom and pop if you feel you were not endowed with a normal size penis. (Now, don't be too hard on mom and pop.) "My penis is too small, too soft, and lacking the endurance to satisfy a friut fly." (Theage.com.au/ Title: Give a man six inches.) Do you feel this way about your little guy? Usually at most, the average size of a erect penis is 6 inches in length and 4.84 inches in girth. Approximately 60% of men do posses this size. (This figure was taken from an independent survey over the course of 3 years and over 350,000 men participants worldwide. There are other numerous reports about a man's average penis size. Some have the average smaller, some larger.) Another study done by a leading condom manufacturer has said that at least 68% of women are unhappy with their partner's penis size. Some of these women did say that size does definitely matter. Well I will leave it up to you men to make your conclusion here on this one. Well in any event, there are many resources available that will help you to achieve a 7 or 8 inch penis if this is what you may think about doing. This task is not a difficult one at all. You would most definitely give your partner more to love. Now if you are deciding to go a step further with this, then you will need a product that is going to Lengthen and Thicken and this will enlarge your penis. Exceed Your Sexuality. A penis size of 7 or 8 inches and a thick girth of no more than 6 inches will add alot more pleasure to your sexual enjoyment. And this will be enough length and girth to keep you and your partner happy for many years to come. And do not go beyond this in trying to lengthen your penis further. You want sex to be quite comfortable with your partner don't you? The 7 or 8 inch penis will be just right. Now if you are already a well skilled player and your partner is satisfied with your 5 or 6 inch penis, then that is a great thing to hear my man. For those who want to add on a few more inches, there is more info available over at my blog to get you started. penis enhancement surgery picture truth about penile enlargement free pnis enlargement exercise real penis enlargement penis enlargement video medical penis enlagement penis enargement vimax top rated penis enlargement pills penis enlargment surgery photo

Since the early 1990s, teen pregnancies across America declined dramatically. However, it is a teen pregnancy fact that 34% of teenage girls in America still get pregnant before they turn the age of 20. This means that America has one of the highest teen pregnancy rates in the world. As does England; in which one in every five births in the country is from a teenager. These are shocking statistics and adults and teenagers alike should be active in teen pregnancy prevention. Talking about sex and pregnancy When a child reaches the age of about 13, parents should be active in approaching their child about their thoughts and ideas of sex and the possible consequences of the act. Although it can initially be embarrassing for both parties, it will pay off in the long run as education and knowledge is the best way to prevent teen pregnancy. Talk to your child about the sexually transmitted diseases and the risk of pregnancy as well as pregnancy prevent methods such as abstinence and contraception. Abstinence The safest path towards teen pregnancy prevention is abstinence. Not having sexual intercourse will mean that you will never have to worry about catching any diseases or unwanted pregnancies which will ultimately change your entire life. Though there will undoubtedly be a lot of peer pressure to have sex, abstaining from sex now will only mean that you will be 100% ready when you do decide to take that step. Contraception The two most popular forms of teen pregnancy prevention come in the form of the condom and the pill. The condom is a rubber latex glove that is placed over the penis during sex to prevent semen from entering the vagina. The condom will not only protect you from possible diseases that can be transmitted through the semen but also from teen pregnancy. Teen pregnancy facts suggest that there are also a large number of teens now taking the birth control pill, and this number is increasing yearly. The birth control pill is taken every day and releases hormones in the body to stop your body from ovulating. If you don't ovulate, then no egg will be released and therefore there will be no egg for the sperm to fertilize. If it is taken every day, the pill is very reliable in terms of pregnancy prevention. However, the pill does not stop you from catching sexually transmitted diseases and infections so you must still be careful. The media often portrays teens regularly having sex. But you don't need to buy into this. The media isn't real and it is perfectly normal to wait until you are ready or for the right person to come along before having sex. The only 100% way to prevent teen pregnancy from occurring is to not have sex at all. enargement free penis pills sample penile enlargement technique penis elargement excersizes natural pennis enlargement exercise natural penis elargement pills vimax natural penis enlargement exercise penis enlarement patch penis enlagement traction device penis enlargment surgery photo

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. permanent pennis enlargement penis enlargment procedure prosolution pennis enlargement pills natural penis enhancement pills pennis enlargement picture natural penile enlargment technique penis enargement cream natural pnis enlargement pills penis enlargment surgery photo

Self-confidence and satisfaction about your physical appearance affect the way you perceive yourself and how you interact with others. You may feel self-conscious of your size, shape or how you look in clothing. Have you ever considered breast augmentation? Would changing your bust size give you a boost? Many women have found that by restoring or creating fullness and shape in their breasts, enhancement of self-esteem, sense of well being and femininity can be achieved. In 2000, over 150,000 women had breast augmentation in the United States. If you are considering having this procedure done, you need to understand the various options regarding the type of implant, the incision site and the placement of the implant. Most women have breast augmentation because they desire enlargement of their breasts. After pregnancy, some women feel their breast size is too small; they may request a breast lift or balancing the different sizes of their breasts. Regardless of why you want the procedure done, your choice to have breast augmentation is a personal one. It is a choice that only you can make. Get the facts and see if breast augmentation is tight for you. Choose an experienced plastic surgeon who is certified by the American Board of Plastic Surgery. The number of breast augmentation surgeries that the plastic surgeon has performed should be considered. If you have friends or colleagues who have had breast augmentation, they might recommend a qualified physician. Contacting the American Society of Plastic Surgeons at their website at www.plasticsurgery.org will provide you with information on qualified surgeons in your area. At your consultation, your surgeon will provide you with various surgical suggestions on what current techniques are available. Your body profile will be reviewed and discussed, as well as the present size and shape of your breasts and what size you wish to have, in order to select the best enhancement for you. There are several specific questions you may wish to ask your plastic surgeon concerning: incision sites; whether you are a candidate for saline or silicone implants, and whether implant placement should be above or below the muscle. The three most common incision areas are; (1) Periareolar - around the dark skin surrounding the nipple; (2) Transaxillary - the armpit area; and (3) Inframammary - where the breast meets the chest. Saline implants are filled by injecting salt water (saline) from a syringe into tubing connected to the implant. The tubing is disconnected after it is filled to the appropriate size, sealing the implant and then removed from your body Determining the correct implant size before surgery is very important. “Trying on” implants filled with various amounts of saline and observing the result with the implants under clothing is very effective. This is an excellent way for the patient to communicate her desired size to her surgeon. Breast augmentation can be a very rewarding experience physically and emotionally. Be in absolute agreement with your surgeon on what look and size you expect. To contact our office call 760-753-6411. Dr. Flynn is a Harvard trained Board Certified Plastic Surgeon who specializes in Botox and other cosmetic procedures. He has been conveniently located in Encinitas for over ten years.